American Journal of Obstetrics & Gynecology
Volume 202, Issue 5 , Pages 464.e1-464.e6 , May 2010

Nicotine restores endothelial dysfunction caused by excess sFlt1 and sEng in an in vitro model of preeclamptic vascular endothelium: a possible therapeutic role of nicotinic acetylcholine receptor (nAChR) agonists for preeclampsia

Received 31 August 2009 ,Revised 20 November 2009 ,Accepted 15 January 2010.

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 Cite this article as: Mimura K, Tomimatsu T, Sharentuya N, et al. Nicotine restores endothelial dysfunction caused by excess sFlt1 and sEng in an in vitro model of preeclamptic vascular endothelium: a possible therapeutic role of nicotinic acetylcholine receptor (nAChR) agonists for preeclampsia. Am J Obstet Gynecol 2010;202:464.e1-6.

 Reprints not available from the authors.

 This study was supported by Grant in Aid for Scientific Research (nos. 19390429, 19599012) from the Ministry of Education, Science, and Culture of Japan.

PII: S0002-9378(10)00067-0

doi: 10.1016/j.ajog.2010.01.037

American Journal of Obstetrics & Gynecology
Volume 202, Issue 5 , Pages 464.e1-464.e6 , May 2010

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