American Journal of Obstetrics & Gynecology
Volume 195, Issue 2 , Pages 522-527 , August 2006

Prenatal alcohol exposure alters GABAAα5 expression: A mechanism of alcohol-induced learning dysfunction

  • Laura Toso, MD

      Affiliations

    • Unit on Perinatal and Developmental Neurobiology, National Institute of Child and Human Development
    • National Institue of Alcohol Abuse and Alcoholism, National Institute of Health, Bethesda, MD
    • Corresponding Author InformationReprint requests: Laura Toso, MD, UPDN, NICHD, NIH and NIAAA, NIH, Blg. 9 Room 1W125, 9 Memorial Drive MSC 0925, Bethesda, MD 20892-0925.
    • ,
  • Robin Roberson, BA

      Affiliations

    • Unit on Perinatal and Developmental Neurobiology, National Institute of Child and Human Development
    • ,
  • Jade Woodard, BS

      Affiliations

    • Unit on Perinatal and Developmental Neurobiology, National Institute of Child and Human Development
    • ,
  • Daniel Abebe, MS

      Affiliations

    • Unit on Perinatal and Developmental Neurobiology, National Institute of Child and Human Development
    • ,
  • Catherine Y. Spong, MD

      Affiliations

    • Unit on Perinatal and Developmental Neurobiology, National Institute of Child and Human Development

Received 7 December 2005 ,Revised 20 January 2006 ,Accepted 24 January 2006.

References 

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  2. Webster WS, Walsh DA, Lipson AH, McEwen SE. Teratogenesis after acute alcohol exposure in inbred and out bred mice. Neurobehav Toxicol. 1980;2:227–234
  3. Spong CY, Abebe D, Gozes I, Brenneman D, Hill J. Prevention of fetal demise and growth restriction in a mouse model of fetal alcohol syndrome. J Pharmacol Exp Ther. 2001;297:774–779
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 Supported by the Division of Intramural Research of National Institute of Child Health and Human Development.

PII: S0002-9378(06)00168-2

doi: 10.1016/j.ajog.2006.01.098

American Journal of Obstetrics & Gynecology
Volume 195, Issue 2 , Pages 522-527 , August 2006

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